Fri Aug 28 05:23:04 SGT 2015  
SINGAPORE
SLIMMING™
    Slimming, Singapore (SG)

Slimming, Singapore (SG)

Summary

Slimming, Singapore (SG) @singaporeslimming_com: Medical slimming, weight/fat loss/management/reduction, diet program/medication clinic, Singapore

Description

Join our weight loss program to manage your obesity. For effective slimming and weight management. Choose our weight loss clinic. The weight management clinic with the program that is most likely able to help you achieve your goals.

Aesthetic services available:

Advertisement: Come to sunny Singapore to have your testing and treatment. Singapore Ministry of Health registered general practice (GP) clinic:
SHIM CLINIC
168 Bedok South Avenue 3 #01-473
Singapore 460168
Tel: (+65) 6446 7446
Fax: (+65) 6449 7446
24hr Answering Tel: (+65) 6333 5550
Web: Slimming, Singapore (SG)
Opening Hours
Monday to Friday: 9 am to 3 pm, 7 pm to 11 pm
Saturday & Sunday: 7 pm to 11 pm
Public Holidays: Closed
Last registration: one hour before closing time.
Walk-in clinic. Appointments not required.
Bring NRIC, Work Pass or Passport for registration.

References


Latest News

Heart Autonomic Modulation in the Obese Children: Evaluation using linear and nonlinear methods
Wed, 26 Aug 2015 22:31:16 +0100 | Autonomic Neuroscience: Basic and Clinical
Background: Functional alterations of the Autonomic Nervous System (ANS) may be due to obesity, but may be identified by the analysis of heart rate variability (HRV). Better understanding of organic systems complexity enables an extension to more sophisticated analysis of HRV, those related to Chaos Theory included, and might be used in the investigation of obese children ANS. Aim: Investigation of autonomic heart modulation in obese children (OC). Methods: The sample consisted of 12 OC (BMI>p97) and 12 eutrophic children (BMI (Source: Autonomic Neuroscience: Basic and Clinical)

Trans-generational effect of maternal obesity on the programming of hypertension: Altered leptin signalling pathway in the central nervous system
Wed, 26 Aug 2015 22:30:38 +0100 | Autonomic Neuroscience: Basic and Clinical
The prevalence of obesity in women among child baring age is increasing and this has been parallel to the increase in obesity in general population around the world. We investigated the trans-generational ‘programming’ of leptin signalling in the central nervous system (CNS) to increase blood pressure (BP), heart rate (HR) and renal sympathetic nerve activity (RSNA) following a high fat diet (HFD)feeding in mothers. Female New Zealand White rabbits were fed a high fat (13%) diet (mHFD) or a control diet (mCD) prior mating and during pregnancy. (Source: Autonomic Neuroscience: Basic and Clinical)

Obstructive sleep apnea syndrome in non severely obese patients with nonalcoholic fatty liver disease
Wed, 26 Aug 2015 22:29:49 +0100 | Autonomic Neuroscience: Basic and Clinical
Background. A high prevalence of obstructive sleep apnea syndrome (OSAS) has been reported in severely obese patients with nonalcoholic fatty liver disease (NAFLD), associated with obesity and metabolic syndrome. Aim of the study is to assess the prevalence of OSAS in patients with NAFLD without severe obesity. Methods. Thirty-five patients with histological NAFLD and body mass index (BMI) (Source: Autonomic Neuroscience: Basic and Clinical)

Seasonal differences in saliva melatonin concentrations and heart rate variability during sleep in obese men
Wed, 26 Aug 2015 22:29:47 +0100 | Autonomic Neuroscience: Basic and Clinical
During the past several decades, obesity has been increasing globally. In Japan, obesity is defined by a BMI of 25kg/m2 or over; 28.6 % of men and 20.6 % of women are obese. Obese people have an increased incidence of developing cardiovascular, renal, and hormonal diseases and sleep disorders. Obese people also have shortened sleep durations. We investigated seasonal differences in melatonin concentrations, heart rates, and heart rate variability during sleep in obese subjects in Japan. Five obese (BMI, 32.0±4.9kg/m2) and five non-obese (BMI, 23.2±2.9kg/m2) men participated in this study in the summer and winter. (Source: Autonomic Neuroscience: Basic and Clinical)

Orthostatic Intolerance and Autonomic Dysfunction following Bariatric Surgery: A case series and review of the literature
Wed, 26 Aug 2015 22:29:17 +0100 | Autonomic Neuroscience: Basic and Clinical
We describe 13 patients referred in the last 5years with orthostatic intolerance, who had undergone bariatric surgery. (Source: Autonomic Neuroscience: Basic and Clinical)

Role of gut to brain signaling in the development of diet-induced obesity
Wed, 26 Aug 2015 22:28:53 +0100 | Autonomic Neuroscience: Basic and Clinical
The vagus nerve connects peripheral organs with the central nervous system. Vagal afferent neurons, located in the nodose ganglia, have processes terminating within the lamina propria of the gut. These neurons express numerous receptors that sense hormones released from the gut epithelium, and provide information about the quantity and type of nutrients throughout the gut to the brain. There is evidence from surgical and chemical lesioning studies, that vagal signaling from the gut to the brain is involved in short-term control of food intake. (Source: Autonomic Neuroscience: Basic and Clinical)

Consumption of a high fat diet inhibits sympathetic outflow to brown adipose tissue (BAT) via vagal afferent activation of neurons in the Nucleus Tractus Solitarius (NTS)
Wed, 26 Aug 2015 22:28:44 +0100 | Autonomic Neuroscience: Basic and Clinical
The recognition of significant depots of BAT in adult humans, the inverse correlation between active BAT and obesity, and the ability of white adipose to be “converted” to a more “brown-like” phenotype have reinvigorated research in this area. The lack of active BAT in obesity has been interpreted to suggest that obese adults lack significant depots of BAT. This seemingly contradicts the observation in rodent models of diet-induced obesity that BAT (UCP-1 expression) is upregulated. The current studies sought to resolve this contradiction by testing the hypotheses that BAT is upregulated in diet-induced obesity but that the sympathetic drive of this tissue is impaired in animals maintained on a high fat diet. (Source: Autonomic Neuroscience: Basic and Clinical)

Elevated sympathetic tone as an etiological factor of cardiometabolic disease
Wed, 26 Aug 2015 22:28:44 +0100 | Autonomic Neuroscience: Basic and Clinical
The sympathetic nervous system (SNS) plays a key role in both cardiovascular and metabolic regulation hence disturbances in SNS regulation are likely to impact on both cardiovascular and metabolic health. With excess adiposity, in particular when visceral fat accumulation is present, sympathetic activation commonly occurs. The cause and extent of sympathetic activation in obesity may be attributed to factors such as the release of adipokines from the adipose tissue, the existence of sleep apnoea, components of the metabolic syndrome, distribution and type of fat and underlying stress. (Source: Autonomic Neuroscience: Basic and Clinical)

The ventromedial hypothalamus as the origin of aberrant blood pressure and sympathetic regulation in diet induced obesity
Wed, 26 Aug 2015 22:28:43 +0100 | Autonomic Neuroscience: Basic and Clinical
Objective: High fat diet (HFD) induced hypertension in rabbits is neurogenic and due to the central action of leptin. This action is dependent on secondary neuronal activation in alpha-melanocortin stimulating hormone (alpha-MSH) and neuropeptide Y (NPY) positive cells. Neurons in the ventromedial hypothalamus (VMH) and Dorsomedial hypothalamus (DMH) are innervated by both neuronal populations and transduce leptin signaling from the Arcuate to other hypothalamic and hindbrain nuclei. The VMH and DMH are also capable of responding to leptin signals directly, independent of NPY or alpha-MSH neurons. (Source: Autonomic Neuroscience: Basic and Clinical)

Variation in appetitive behaviour between pet dogs; association with obesity and relationship to owner management factors.
Wed, 26 Aug 2015 20:26:56 +0100 | Journal of Veterinary Behavior: Clinical Applications and Research
Dog obesity is an increasing problem with significant associated morbidity. Previous research has focussed on owner management but individual variation in dog appetitive behaviour is likely to contribute to breed and individual predisposition to obesity. To investigate appetitive behaviour in dogs and its relationship with owner management. The DORA questionnaire is a reliable, informative owner-reported measure of canine eating behaviour, owner management and health factors which can affect the development of obesity. (Source: Journal of Veterinary Behavior: Clinical Applications and Research)