Wed Nov 26 18:56:10 SGT 2014  
SINGAPORE
SLIMMING™
    Jaw slimming, Singapore (SG)

Jaw slimming, Singapore (SG)

Summary

Jaw slimming, Singapore (SG) @singaporeslimming_com: Medical slimming, weight/fat loss/management/reduction, diet program/medication clinic, Singapore

Description

Join our weight loss program to manage your obesity. For effective slimming and weight management. Choose our weight loss clinic. The weight management clinic with the program that is most likely able to help you achieve your goals.

Aesthetic services available:

Advertisement: Come to sunny Singapore to have your testing and treatment. Singapore Ministry of Health registered general practice (GP) clinic:
SHIM CLINIC
168 Bedok South Avenue 3 #01-473
Singapore 460168
Tel: (+65) 6446 7446
Fax: (+65) 6449 7446
24hr Answering Tel: (+65) 6333 5550
Web: Jaw slimming, Singapore (SG)
Opening Hours
Monday to Friday: 9 am to 3 pm, 7 pm to 11 pm
Saturday & Sunday: 7 pm to 11 pm
Public Holidays: Closed
Last registration: one hour before closing time.
Walk-in clinic. Appointments not required.
Bring NRIC, Work Pass or Passport for registration.

References


Latest News

Weight Loss Maintenance for 2 Years after a 6-Month Randomised Controlled Trial Comparing Education-Only and Group-Based Support in Japanese Adults
Tue, 25 Nov 2014 18:11:13 +0100 | Obesity Facts
Conclusion: The effects of group-based support disappear within 2 years. Increasing physical activity may be a crucial factor for successful maintenance of weight loss.Obes Facts 2014;7:376-387 (Source: Obesity Facts)

Parental Characteristics Have a Larger Effect on Children's Health Behaviour than Their Body Weight
Tue, 25 Nov 2014 18:11:13 +0100 | Obesity Facts
Conclusion: These results suggest that parental behaviour and role modelling provide an important contribution to childrens' health behaviour, especially at younger ages.Obes Facts 2014;7:388-398 (Source: Obesity Facts)

Serum Leptin and Skeletal Differences between Obese and Non-Obese Patients with Chronic Obstructive Pulmonary Disease
Tue, 25 Nov 2014 18:11:13 +0100 | Obesity Facts
Conclusion: A positive relationship between FMI and BMD was found only in non-obese but not in obese COPD patients. Leptin concentration was associated positively with the total hip T score only in obese COPD patients, suggesting its protective role on the skeleton of obese COPD patients.Obes Facts 2014;7:399-407 (Source: Obesity Facts)

Investigating correlations in the altered metabolic profiles of obese and diabetic subjects in a South Indian Asian population using an NMR-based metabolomic approach
Tue, 25 Nov 2014 15:11:42 +0100 | RSC - Mol. BioSyst. latest articles
Mol. BioSyst., 2014, Accepted ManuscriptDOI: 10.1039/C4MB00507D, PaperNavdeep Gogna, Murahari K, Anup Mammen Oomen, Kavita DoraiIt is well known that obesity/high body mass index (BMI) plays a key role in the evolution of insulin resistance and Type-2 diabetes mellitus. However, the exact mechanism underlying its...The content of this RSS Feed (c) The Royal Society of Chemistry (Source: RSC - Mol. BioSyst. latest articles)

Correlation of Circulating Acid-Labile Subunit Levels with Insulin Sensitivity and Serum LDL Cholesterol in Patients with Type 2 Diabetes: Findings from a Prospective Study with Rosiglitazone.
Tue, 25 Nov 2014 11:31:27 +0100 | PPAR Research
This study suggested a BMI-dependent effect of RSG treatment on ALS levels. Reduction of ALS by RSG increases the risk of atherosclerosis in individuals with type 2 diabetes.

Radix Astragali Improves Dysregulated Triglyceride Metabolism and Attenuates Macrophage Infiltration in Adipose Tissue in High-Fat Diet-Induced Obese Male Rats through Activating mTORC1-PPAR γ Signaling Pathway.
Tue, 25 Nov 2014 11:31:27 +0100 | PPAR Research
In conclusion, the mTORC1-PPAR γ signaling pathway is a potential mechanism through which RA exerts beneficial effects on the disturbance of triglyceride metabolism and dysfunction of adipose tissue in DIO rats.

Effects and Potential Mechanisms of Pioglitazone on Lipid Metabolism in Obese Diabetic KKAy Mice.
Tue, 25 Nov 2014 11:31:27 +0100 | PPAR Research
This study aimed to analyze the effects and potential mechanisms of pioglitazone on triglyceride and cholesterol metabolism in obese diabetic KKAy mice. Pioglitazone was orally administered to KKAy mice over 30 days. Compared to C57BL/6J mice, KKAy mice developed obvious insulin resistance, hepatic steatosis, and hyperlipidemia. Pioglitazone treatment resulted in deteriorated microvesicular steatosis and elevated hepatic triglyceride levels, though plasma triglyceride and free fatty acid levels were reduced by the treatment, compared to nontreated KKAy mice. Plasma alanine aminotransferase activities were also significantly increased. Additionally, pioglitazone increased plasma concentrations of total cholesterol, HDL-cholesterol, and LDL-cholesterol but decreased hepatic cholesterol. Gene...

Pseudoginsenoside F11, a Novel Partial PPAR γ Agonist, Promotes Adiponectin Oligomerization and Secretion in 3T3-L1 Adipocytes.
Tue, 25 Nov 2014 11:31:27 +0100 | PPAR Research
In this study, we found that p-F11 activates PPAR γ with modest adipogenic activity. In addition, p-F11 promotes adiponectin oligomerization and secretion in 3T3-L1 adipocytes. We also found that p-F11 inhibits obesity-linked phosphorylation of PPAR γ at Ser-273 by Cdk5. Therefore, p-F11 is a novel partial PPAR γ agonist, which might have the potential to be developed as a new PPAR γ -targeted therapeutics for type 2 diabetes.

The PPAR α / γ Agonist, Tesaglitazar, Improves Insulin Mediated Switching of Tissue Glucose and Free Fatty Acid Utilization In Vivo in the Obese Zucker Rat.
Tue, 25 Nov 2014 11:31:27 +0100 | PPAR Research
In conclusion, the results demonstrate that tesaglitazar has a remarkable ability to improve insulin mediated control of glucose and FFA fluxes in obese Zucker rats.

Does Pro(12)Ala Polymorphism Enhance the Physiological Role of PPARγ2?
Tue, 25 Nov 2014 11:31:27 +0100 | PPAR Research
Authors: Pereira AC, Oliveira R, Castro AC, Fernandes R